COVID-19 infection is associated with thromboembolism affecting cerebral veins and venous sinuses, propitiating diagnostic challenges with more frequent etiologies 1-6) Venous thromboembolism has been observed in 7.7% to 28.0% of the hospitalized patients with diagnosis of COVID-19 in spite of utilizing the appropriate routine prophylaxis 2 . Cytokine storm and endothelial lesions due to the infection can cause hypercoagulation, deep venous and cerebral venous sinus thrombosis (cVST), or pulmonary embolism 1,4,6. The diagnostic suspicion among health workers about the hypothesis of cVST should be enhanced because this entity can be misdiagnosed, underdiagnosed, or under- -reported 1,3. The clinical hypothesis is confirmed by laboratory tests and studies of images by computed tomography (CT), magnetic resonance imaging (MRI), and angiography procedures; and the patients often successfully improve after using heparin and oral anticoagulation 1-6.
